BEGIN:VCALENDAR
VERSION:2.0
PRODID:-//Institute for Bioengineering of Catalonia - ECPv6.15.18//NONSGML v1.0//EN
CALSCALE:GREGORIAN
METHOD:PUBLISH
X-WR-CALNAME:Institute for Bioengineering of Catalonia
X-ORIGINAL-URL:https://ibecbarcelona.eu
X-WR-CALDESC:Events for Institute for Bioengineering of Catalonia
REFRESH-INTERVAL;VALUE=DURATION:PT1H
X-Robots-Tag:noindex
X-PUBLISHED-TTL:PT1H
BEGIN:VTIMEZONE
TZID:Europe/Madrid
BEGIN:DAYLIGHT
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
TZNAME:CEST
DTSTART:20160327T010000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
TZNAME:CET
DTSTART:20161030T010000
END:STANDARD
BEGIN:DAYLIGHT
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
TZNAME:CEST
DTSTART:20170326T010000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
TZNAME:CET
DTSTART:20171029T010000
END:STANDARD
BEGIN:DAYLIGHT
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
TZNAME:CEST
DTSTART:20180325T010000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
TZNAME:CET
DTSTART:20181028T010000
END:STANDARD
END:VTIMEZONE
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T120000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170403T100450Z
LAST-MODIFIED:20170403T100450Z
UID:96030-1493294400-1493298000@ibecbarcelona.eu
SUMMARY:IBEC-IRB Barcelona joint seminar: Erik Sahai
DESCRIPTION:Imaging therapy response and failure\nErik Sahai\, The Francis Crick Institute\, London\nMany tumors show an initial response to targeted therapies before genetic resistance emerges\, however little is known about how tumor cells tolerate therapy before genetic resistance dominates. We present data that shows how the ECM generates a ‘safe haven’ in which melanoma cells can tolerate targeted therapy.  This supports the population of cancer cells from which genetically resistance emerges. We are now studying the emergence of resistant clones and clonal competition in the context of ‘safe havens’. Finally\, we present analysis of organ specific responses to targeted therapy in melanoma.
URL:https://ibecbarcelona.eu/event/ibec-irb-barcelona-joint-seminar-erik-sahai-2/
LOCATION:Sala Félix Serratossa\, Parc Científic de Barcelona\, Barcelona\, Spain
CATEGORIES:Joint seminar / workshop / symposium
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T120000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170403T100450Z
LAST-MODIFIED:20170403T100450Z
UID:96031-1493294400-1493298000@ibecbarcelona.eu
SUMMARY:IBEC-IRB Barcelona joint seminar: Erik Sahai
DESCRIPTION:Imaging therapy response and failure\nErik Sahai\, The Francis Crick Institute\, London\nMany tumors show an initial response to targeted therapies before genetic resistance emerges\, however little is known about how tumor cells tolerate therapy before genetic resistance dominates. We present data that shows how the ECM generates a ‘safe haven’ in which melanoma cells can tolerate targeted therapy.  This supports the population of cancer cells from which genetically resistance emerges. We are now studying the emergence of resistant clones and clonal competition in the context of ‘safe havens’. Finally\, we present analysis of organ specific responses to targeted therapy in melanoma.
URL:https://ibecbarcelona.eu/event/ibec-irb-barcelona-joint-seminar-erik-sahai-3/
LOCATION:Sala Félix Serratossa\, Parc Científic de Barcelona\, Barcelona\, Spain
CATEGORIES:Joint seminar / workshop / symposium
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T110000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170425T112649Z
LAST-MODIFIED:20170425T112833Z
UID:28860-1493290800-1493298000@ibecbarcelona.eu
SUMMARY:PhD Thesis Defence: Anna Crespo
DESCRIPTION:“Estudi transcripcional i funcional de les ribonucleotidil reductases de Pseudomonas aeruginosa“\nAnna Crespo\, Bacterial infections and antimicrobial therapies group\nAnna will be defending her PhD thesis on Thursday 27th April at 11:00 in the Aula Magna of the UB’s Facultat de Farmàcia i Ciències de l’Alimentació. \nEverybody is welcome to attend. \n—\nIf you’re an IBEC PhD student and would like to advertise your PhD defence on the IBEC calendar\, please contact vleigh@ibecbarcelona.eu
URL:https://ibecbarcelona.eu/event/phd-thesis-defence-anna-crespo/
LOCATION:Aula Magna\, Faculty of Pharmacy\, Av. Joan XXIII s/n\, Barcelona\, Spain
CATEGORIES:PhD Thesis Defence
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T110000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170425T112649Z
LAST-MODIFIED:20170425T112649Z
UID:96042-1493290800-1493298000@ibecbarcelona.eu
SUMMARY:PhD Thesis Defence: Anna Crespo
DESCRIPTION:“Estudi transcripcional i funcional de les ribonucleotidil reductases de Pseudomonas aeruginosa“\nAnna Crespo\, Bacterial infections and antimicrobial therapies group\nAnna will be defending her PhD thesis on Thursday 27th April at 11:00 in the Aula Magna of the UB’s Facultat de Farmàcia i Ciències de l’Alimentació. \nEverybody is welcome to attend. \n—\nIf you’re an IBEC PhD student and would like to advertise your PhD defence on the IBEC calendar\, please contact vleigh@ibecbarcelona.eu
URL:https://ibecbarcelona.eu/event/phd-thesis-defence-anna-crespo-2/
LOCATION:Aula Magna\, Faculty of Pharmacy\, Av. Joan XXIII s/n\, Barcelona\, Spain
CATEGORIES:PhD Thesis Defence
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T110000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170425T112649Z
LAST-MODIFIED:20170425T112649Z
UID:96043-1493290800-1493298000@ibecbarcelona.eu
SUMMARY:PhD Thesis Defence: Anna Crespo
DESCRIPTION:“Estudi transcripcional i funcional de les ribonucleotidil reductases de Pseudomonas aeruginosa“\nAnna Crespo\, Bacterial infections and antimicrobial therapies group\nAnna will be defending her PhD thesis on Thursday 27th April at 11:00 in the Aula Magna of the UB’s Facultat de Farmàcia i Ciències de l’Alimentació. \nEverybody is welcome to attend. \n—\nIf you’re an IBEC PhD student and would like to advertise your PhD defence on the IBEC calendar\, please contact vleigh@ibecbarcelona.eu
URL:https://ibecbarcelona.eu/event/phd-thesis-defence-anna-crespo-3/
LOCATION:Aula Magna\, Faculty of Pharmacy\, Av. Joan XXIII s/n\, Barcelona\, Spain
CATEGORIES:PhD Thesis Defence
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170427T110000
DTEND;TZID=Europe/Madrid:20170427T130000
DTSTAMP:20260406T000101
CREATED:20170425T112649Z
LAST-MODIFIED:20170425T112649Z
UID:96044-1493290800-1493298000@ibecbarcelona.eu
SUMMARY:PhD Thesis Defence: Anna Crespo
DESCRIPTION:“Estudi transcripcional i funcional de les ribonucleotidil reductases de Pseudomonas aeruginosa“\nAnna Crespo\, Bacterial infections and antimicrobial therapies group\nAnna will be defending her PhD thesis on Thursday 27th April at 11:00 in the Aula Magna of the UB’s Facultat de Farmàcia i Ciències de l’Alimentació. \nEverybody is welcome to attend. \n—\nIf you’re an IBEC PhD student and would like to advertise your PhD defence on the IBEC calendar\, please contact vleigh@ibecbarcelona.eu
URL:https://ibecbarcelona.eu/event/phd-thesis-defence-anna-crespo-4/
LOCATION:Aula Magna\, Faculty of Pharmacy\, Av. Joan XXIII s/n\, Barcelona\, Spain
CATEGORIES:PhD Thesis Defence
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170426T090000
DTEND;TZID=Europe/Madrid:20170426T130000
DTSTAMP:20260406T000101
CREATED:20170327T094639Z
LAST-MODIFIED:20170327T094639Z
UID:96029-1493197200-1493211600@ibecbarcelona.eu
SUMMARY:Curso de Primeros Auxilios
DESCRIPTION:El objetivo del curso es ser una ayuda para adquirir los conocimientos necesarios de cara a prestar los primeros auxilios a aquellas personas que sufran un accidente o enfermedad repentina\, hasta la llegada del personal sanitario. De esta primera actuación va a depender\, en gran medida\, el estado general y la posterior evolución de la persona. Con este curso conoceremos los tratamientos que están a nuestro alcance y sabremos lo que NO debemos hacer nunca para no agravar el estado del lesionado. \nFechas: \n26 de Abril. 4 horas\, de 9 a 13h. \nParticipantes: \nToda la comunidad IBEC
URL:https://ibecbarcelona.eu/event/curso-de-primeros-auxilios-3/
CATEGORIES:Professional and Personal Development
ORGANIZER;CN="IBEC":MAILTO:www.ibecbarcelona.eu
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170426T090000
DTEND;TZID=Europe/Madrid:20170426T130000
DTSTAMP:20260406T000101
CREATED:20170327T094639Z
LAST-MODIFIED:20170327T094639Z
UID:28694-1493197200-1493211600@ibecbarcelona.eu
SUMMARY:Curso de Primeros Auxilios
DESCRIPTION:El objetivo del curso es ser una ayuda para adquirir los conocimientos necesarios de cara a prestar los primeros auxilios a aquellas personas que sufran un accidente o enfermedad repentina\, hasta la llegada del personal sanitario. De esta primera actuación va a depender\, en gran medida\, el estado general y la posterior evolución de la persona. Con este curso conoceremos los tratamientos que están a nuestro alcance y sabremos lo que NO debemos hacer nunca para no agravar el estado del lesionado. \nFechas: \n26 de Abril. 4 horas\, de 9 a 13h. \nParticipantes: \nToda la comunidad IBEC
URL:https://ibecbarcelona.eu/event/curso-de-primeros-auxilios/
CATEGORIES:Professional and Personal Development
ORGANIZER;CN="IBEC":MAILTO:www.ibecbarcelona.eu
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170426T090000
DTEND;TZID=Europe/Madrid:20170426T130000
DTSTAMP:20260406T000101
CREATED:20170324T111018Z
LAST-MODIFIED:20170324T111018Z
UID:96028-1493197200-1493211600@ibecbarcelona.eu
SUMMARY:Curso de primeros auxilios
DESCRIPTION:El objetivo del curso es ser una ayuda para adquirir los conocimientos necesarios de cara a prestar los primeros auxilios a aquellas personas que sufran un accidente o enfermedad repentina\, hasta la llegada del personal sanitario. De esta primera actuación va a depender\, en gran medida\, el estado general y la posterior evolución de la persona. Con este curso conoceremos los tratamientos que están a nuestro alcance y sabremos lo que NO debemos hacer nunca para no agravar el estado del lesionado. \nFechas: \n26 de Abril. 4 horas\, de 9 a 13h. \nParticipantes: \nToda la comunidad IBEC \nFormador: ASEM.
URL:https://ibecbarcelona.eu/event/curso-de-primeros-auxilios-2/
CATEGORIES:Professional and Personal Development
END:VEVENT
BEGIN:VEVENT
DTSTART;VALUE=DATE:20170425
DTEND;VALUE=DATE:20170427
DTSTAMP:20260406T000101
CREATED:20170404T102254Z
LAST-MODIFIED:20170801T105133Z
UID:28771-1493078400-1493251199@ibecbarcelona.eu
SUMMARY:II Festival de la Nanociencia (10alamenos9)
DESCRIPTION:IBEC is an organiser of this year’s Festival de la Nanociencia (10alamenos9)\, funded by FECYT.\nThe festival will take place simultaneously in Barcelona\, Saragossa\, Bellaterra\, San Sebastian and Madrid. \nThe programme will include seminars by Gabriel Gomila and Samuel Sanchez and a Workshop on Drug Delivery (Cosmocaixa 26/04/2017)\, visits to IBEC and workshop for students (March and April at IBEC)\, and a Nano Workshop in collaborations with Il·lustraciència (Cosmocaixa 25/04/2017 and 26/04/2017). \nMore details here.
URL:https://ibecbarcelona.eu/event/ii-festival-de-la-nanociencia-10alamenos9/
CATEGORIES:Outreach / Fair / Festival
END:VEVENT
BEGIN:VEVENT
DTSTART;VALUE=DATE:20170425
DTEND;VALUE=DATE:20170427
DTSTAMP:20260406T000101
CREATED:20170404T102254Z
LAST-MODIFIED:20170404T102254Z
UID:96035-1493078400-1493251199@ibecbarcelona.eu
SUMMARY:II Festival de la Nanociencia (10alamenos9)
DESCRIPTION:IBEC is an organiser of this year’s Festival de la Nanociencia (10alamenos9)\, funded by FECYT.\nThe festival will take place simultaneously in Barcelona\, Saragossa\, Bellaterra\, San Sebastian and Madrid. \nThe programme will include seminars by Gabriel Gomila and Samuel Sanchez and a Workshop on Drug Delivery (Cosmocaixa 26/04/2017)\, visits to IBEC and workshop for students (March and April at IBEC)\, and a Nano Workshop in collaborations with Il·lustraciència (Cosmocaixa 25/04/2017 and 26/04/2017). \nMore details here.
URL:https://ibecbarcelona.eu/event/ii-festival-de-la-nanociencia-10alamenos9-2/
CATEGORIES:Outreach / Fair / Festival
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170425T000000
DTEND;TZID=Europe/Madrid:20170426T235959
DTSTAMP:20260406T000101
CREATED:20170404T102254Z
LAST-MODIFIED:20170404T102254Z
UID:96036-1493078400-1493251199@ibecbarcelona.eu
SUMMARY:II Festival de la Nanociencia (10alamenos9)
DESCRIPTION:IBEC is an organiser of this year’s Festival de la Nanociencia (10alamenos9)\, funded by FECYT.\nThe festival will take place simultaneously in Barcelona\, Saragossa\, Bellaterra\, San Sebastian and Madrid. \nThe programme will include seminars by Gabriel Gomila and Samuel Sanchez and a Workshop on Drug Delivery (Cosmocaixa 26/04/2017)\, visits to IBEC and workshop for students (March and April at IBEC)\, and a Nano Workshop in collaborations with Il·lustraciència (Cosmocaixa 25/04/2017 and 26/04/2017). \nMore details here.
URL:https://ibecbarcelona.eu/event/ii-festival-de-la-nanociencia-10alamenos9-3/
CATEGORIES:Outreach / Fair / Festival
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170425T000000
DTEND;TZID=Europe/Madrid:20170426T235959
DTSTAMP:20260406T000101
CREATED:20170404T102254Z
LAST-MODIFIED:20170404T102254Z
UID:96037-1493078400-1493251199@ibecbarcelona.eu
SUMMARY:II Festival de la Nanociencia (10alamenos9)
DESCRIPTION:IBEC is an organiser of this year’s Festival de la Nanociencia (10alamenos9)\, funded by FECYT.\nThe festival will take place simultaneously in Barcelona\, Saragossa\, Bellaterra\, San Sebastian and Madrid. \nThe programme will include seminars by Gabriel Gomila and Samuel Sanchez and a Workshop on Drug Delivery (Cosmocaixa 26/04/2017)\, visits to IBEC and workshop for students (March and April at IBEC)\, and a Nano Workshop in collaborations with Il·lustraciència (Cosmocaixa 25/04/2017 and 26/04/2017). \nMore details here.
URL:https://ibecbarcelona.eu/event/ii-festival-de-la-nanociencia-10alamenos9-4/
CATEGORIES:Outreach / Fair / Festival
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170331T100000
DTEND;TZID=Europe/Madrid:20170331T230000
DTSTAMP:20260406T000101
CREATED:20170202T093241Z
LAST-MODIFIED:20170202T093241Z
UID:95989-1490954400-1491001200@ibecbarcelona.eu
SUMMARY:PhD Discussions Sessions: Aida Garrido and Marina Uroz
DESCRIPTION:Optical control of endogenous receptors and cellular excitability using targeted covalent photoswitches\nAida Garrido\, Nanoprobes and nanoswitches group\nLight-regulated drugs allow remotely photoswitching biological activity and enable plausible therapies based on small molecules. However\, only freely diffusible photochromic ligands have been shown to work directly in endogenous receptors and methods for covalent attachment depend on genetic manipulation. Here we introduce a chemical strategy to covalently conjugate and photoswitch the activity of endogenous proteins and demonstrate its application to the kainate receptor channel GluK1. The approach is based on photoswitchable ligands containing a short-lived\, highly reactive anchoring group that is targeted at the protein of interest by ligand affinity. These targeted covalent photoswitches (TCPs) constitute a new class of light-regulated drugs and act as prosthetic molecules that photocontrol the activity of GluK1-expressing neurons\, and restore photoresponses in degenerated retina. The modularity of TCPs enables the application to different ligands and opens the way to new therapeutic opportunities. \n  \nTraction forces at the cytokinetic ring regulate cell division and polyploidy in the migrating zebrafish epicardium\nMarina Uroz\, Integrative Cell and Tissue Dynamics group\nEpithelial repair and regeneration are driven by collective cell migration and division. Both cellular functions involve tightly controlled mechanical events. Mechanics of collective cell migration is increasingly well understood\, but physical forces associated with cell division in cohesive epithelia have escaped experimental observation. Using the zebrafish epicardium as a model system\, we show that cells dividing in a migrating epithelium exert large cell-extracellular matrix (ECM) forces during cytokinesis. These forces point towards the midbody and are exerted through paxillin-rich focal adhesions that connect the cytokinetic ring to the underlying extracellular matrix. Large forces at these adhesions are associated with failure of cytokinesis and polyploidy\, indicating that abnormal cell-matrix adhesion at the cleavage furrow impedes the latest stages of abscission. Mechanical interaction between the cytokinetic ring and the ECM thus provide a new mechanism for the regulation of cell division and polyploidy.  \n 
URL:https://ibecbarcelona.eu/event/phd-discussions-sessions-aida-garrido-and-marina-uroz-3/
CATEGORIES:PhD Discussions Session
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170331T100000
DTEND;TZID=Europe/Madrid:20170331T230000
DTSTAMP:20260406T000101
CREATED:20170202T093241Z
LAST-MODIFIED:20170202T093241Z
UID:95990-1490954400-1491001200@ibecbarcelona.eu
SUMMARY:PhD Discussions Sessions: Aida Garrido and Marina Uroz
DESCRIPTION:Optical control of endogenous receptors and cellular excitability using targeted covalent photoswitches\nAida Garrido\, Nanoprobes and nanoswitches group\nLight-regulated drugs allow remotely photoswitching biological activity and enable plausible therapies based on small molecules. However\, only freely diffusible photochromic ligands have been shown to work directly in endogenous receptors and methods for covalent attachment depend on genetic manipulation. Here we introduce a chemical strategy to covalently conjugate and photoswitch the activity of endogenous proteins and demonstrate its application to the kainate receptor channel GluK1. The approach is based on photoswitchable ligands containing a short-lived\, highly reactive anchoring group that is targeted at the protein of interest by ligand affinity. These targeted covalent photoswitches (TCPs) constitute a new class of light-regulated drugs and act as prosthetic molecules that photocontrol the activity of GluK1-expressing neurons\, and restore photoresponses in degenerated retina. The modularity of TCPs enables the application to different ligands and opens the way to new therapeutic opportunities. \n  \nTraction forces at the cytokinetic ring regulate cell division and polyploidy in the migrating zebrafish epicardium\nMarina Uroz\, Integrative Cell and Tissue Dynamics group\nEpithelial repair and regeneration are driven by collective cell migration and division. Both cellular functions involve tightly controlled mechanical events. Mechanics of collective cell migration is increasingly well understood\, but physical forces associated with cell division in cohesive epithelia have escaped experimental observation. Using the zebrafish epicardium as a model system\, we show that cells dividing in a migrating epithelium exert large cell-extracellular matrix (ECM) forces during cytokinesis. These forces point towards the midbody and are exerted through paxillin-rich focal adhesions that connect the cytokinetic ring to the underlying extracellular matrix. Large forces at these adhesions are associated with failure of cytokinesis and polyploidy\, indicating that abnormal cell-matrix adhesion at the cleavage furrow impedes the latest stages of abscission. Mechanical interaction between the cytokinetic ring and the ECM thus provide a new mechanism for the regulation of cell division and polyploidy.  \n 
URL:https://ibecbarcelona.eu/event/phd-discussions-sessions-aida-garrido-and-marina-uroz-4/
CATEGORIES:PhD Discussions Session
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170331T100000
DTEND;TZID=Europe/Madrid:20170331T230000
DTSTAMP:20260406T000101
CREATED:20170202T093241Z
LAST-MODIFIED:20170323T102457Z
UID:27406-1490954400-1491001200@ibecbarcelona.eu
SUMMARY:PhD Discussions Sessions: Aida Garrido and Marina Uroz
DESCRIPTION:Optical control of endogenous receptors and cellular excitability using targeted covalent photoswitches\nAida Garrido\, Nanoprobes and nanoswitches group\nLight-regulated drugs allow remotely photoswitching biological activity and enable plausible therapies based on small molecules. However\, only freely diffusible photochromic ligands have been shown to work directly in endogenous receptors and methods for covalent attachment depend on genetic manipulation. Here we introduce a chemical strategy to covalently conjugate and photoswitch the activity of endogenous proteins and demonstrate its application to the kainate receptor channel GluK1. The approach is based on photoswitchable ligands containing a short-lived\, highly reactive anchoring group that is targeted at the protein of interest by ligand affinity. These targeted covalent photoswitches (TCPs) constitute a new class of light-regulated drugs and act as prosthetic molecules that photocontrol the activity of GluK1-expressing neurons\, and restore photoresponses in degenerated retina. The modularity of TCPs enables the application to different ligands and opens the way to new therapeutic opportunities. \n  \nTraction forces at the cytokinetic ring regulate cell division and polyploidy in the migrating zebrafish epicardium\nMarina Uroz\, Integrative Cell and Tissue Dynamics group\nEpithelial repair and regeneration are driven by collective cell migration and division. Both cellular functions involve tightly controlled mechanical events. Mechanics of collective cell migration is increasingly well understood\, but physical forces associated with cell division in cohesive epithelia have escaped experimental observation. Using the zebrafish epicardium as a model system\, we show that cells dividing in a migrating epithelium exert large cell-extracellular matrix (ECM) forces during cytokinesis. These forces point towards the midbody and are exerted through paxillin-rich focal adhesions that connect the cytokinetic ring to the underlying extracellular matrix. Large forces at these adhesions are associated with failure of cytokinesis and polyploidy\, indicating that abnormal cell-matrix adhesion at the cleavage furrow impedes the latest stages of abscission. Mechanical interaction between the cytokinetic ring and the ECM thus provide a new mechanism for the regulation of cell division and polyploidy.  \n 
URL:https://ibecbarcelona.eu/event/phd-discussions-sessions-aida-garrido-and-marina-uroz/
CATEGORIES:PhD Discussions Session
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170331T100000
DTEND;TZID=Europe/Madrid:20170331T230000
DTSTAMP:20260406T000101
CREATED:20170202T093241Z
LAST-MODIFIED:20170202T093241Z
UID:95983-1490954400-1491001200@ibecbarcelona.eu
SUMMARY:PhD Discussions Sessions: Aida Garrido and Marina Uroz
DESCRIPTION:Optical control of endogenous receptors and cellular excitability using targeted covalent photoswitches\nAida Garrido\, Nanoprobes and nanoswitches group\nLight-regulated drugs allow remotely photoswitching biological activity and enable plausible therapies based on small molecules. However\, only freely diffusible photochromic ligands have been shown to work directly in endogenous receptors and methods for covalent attachment depend on genetic manipulation. Here we introduce a chemical strategy to covalently conjugate and photoswitch the activity of endogenous proteins and demonstrate its application to the kainate receptor channel GluK1. The approach is based on photoswitchable ligands containing a short-lived\, highly reactive anchoring group that is targeted at the protein of interest by ligand affinity. These targeted covalent photoswitches (TCPs) constitute a new class of light-regulated drugs and act as prosthetic molecules that photocontrol the activity of GluK1-expressing neurons\, and restore photoresponses in degenerated retina. The modularity of TCPs enables the application to different ligands and opens the way to new therapeutic opportunities. \n  \nTraction forces at the cytokinetic ring regulate cell division and polyploidy in the migrating zebrafish epicardium\nMarina Uroz\, Integrative Cell and Tissue Dynamics group\nEpithelial repair and regeneration are driven by collective cell migration and division. Both cellular functions involve tightly controlled mechanical events. Mechanics of collective cell migration is increasingly well understood\, but physical forces associated with cell division in cohesive epithelia have escaped experimental observation. Using the zebrafish epicardium as a model system\, we show that cells dividing in a migrating epithelium exert large cell-extracellular matrix (ECM) forces during cytokinesis. These forces point towards the midbody and are exerted through paxillin-rich focal adhesions that connect the cytokinetic ring to the underlying extracellular matrix. Large forces at these adhesions are associated with failure of cytokinesis and polyploidy\, indicating that abnormal cell-matrix adhesion at the cleavage furrow impedes the latest stages of abscission. Mechanical interaction between the cytokinetic ring and the ECM thus provide a new mechanism for the regulation of cell division and polyploidy.  \n 
URL:https://ibecbarcelona.eu/event/phd-discussions-sessions-aida-garrido-and-marina-uroz-2/
CATEGORIES:PhD Discussions Session
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170329T120000
DTEND;TZID=Europe/Madrid:20170329T130000
DTSTAMP:20260406T000101
CREATED:20170308T135727Z
LAST-MODIFIED:20170308T135727Z
UID:96007-1490788800-1490792400@ibecbarcelona.eu
SUMMARY:IBEC Seminar: Aranzazu Villasante
DESCRIPTION:Cancer Engineering: Strategies to Engineer Predictable Tumor Models\nDr. Aranzazu Villasante\, Department of Biomedical Engineering\, Columbia University\, New York\nAlthough many drugs show promise in monolayer or in animal models systems\, most fail to translate in humans and this is because they lack of ability to replicate the human microenvironment in patients. In response to these limitations\, I have generated a set of predictable tissue-engineered (TE) models of cancer by using different strategies. Today\, I am going to focus on some of these approaches to engineer pediatric tumors in vitro. Firstly\, I will show a TE model of Ewing’s sarcoma (ES) within its bone niche. This particular strategy is based on engineered human bone by introducing osteoclasts in co-culture with osteoblasts in the 3-dimensional bone niche. This model mimics bone remodeling and recapitulates some of the features observed in the osteolytic process in cancer and also\, the effects of the therapeutic reagent Zoledronic acid observed in patients. The second strategy consists in designing biomaterials with the same tumor composition to mimic the biological and mechanical properties of tumors from patients. I have developed 3D porous collagen 1-hyaluronic acid scaffolds (Col1-HA scaffolds) for studies of tumor derivedexosomes\, which are known to be initiators of pre-metastatic niche formation in certain sites. Interestingly\, I found high levels of a critical mediator of ES growth and metastasis (EZH2) in exosomes isolated from both patients and TE model of ES. Alternatively\, we cultured TE models based on Col1-HA scaffolds into a mechanical loading bioreactor for better mimicking biomechanical forces in ES. We found that biomechanical stimuli modulate osteolytic-related proteins (i.e. RUNX2) and sensitivity to anticancer drugs\, such as Sorafenib. I will also explain the use of perfusion bioreactors and cell sheet engineering to develop a novel model of Neuroblastoma (NB) to study the effect of consolidative drugs\, such as Isotretinoin\, on tumor vasculature and stem-like cells. Here\, I will show the existence of sub-populations of NB cells with different levels of stemness properties; these levels are related to the capacity of stem-like cells to transdifferentiate and also\, to chemoresistance and relapse. Finally\, the take-home message of my talk will be that TE models can bridge the gap between 2D in vitro cultures and in vivo animal models in a predictive\, inexpensive and low timeconsuming fashion for successfully understand cancer biology and improve cancer treatments.
URL:https://ibecbarcelona.eu/event/ibec-seminar-aranzazu-villasante-2/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170329T120000
DTEND;TZID=Europe/Madrid:20170329T130000
DTSTAMP:20260406T000101
CREATED:20170308T135727Z
LAST-MODIFIED:20170308T135727Z
UID:96009-1490788800-1490792400@ibecbarcelona.eu
SUMMARY:IBEC Seminar: Aranzazu Villasante
DESCRIPTION:Cancer Engineering: Strategies to Engineer Predictable Tumor Models\nDr. Aranzazu Villasante\, Department of Biomedical Engineering\, Columbia University\, New York\nAlthough many drugs show promise in monolayer or in animal models systems\, most fail to translate in humans and this is because they lack of ability to replicate the human microenvironment in patients. In response to these limitations\, I have generated a set of predictable tissue-engineered (TE) models of cancer by using different strategies. Today\, I am going to focus on some of these approaches to engineer pediatric tumors in vitro. Firstly\, I will show a TE model of Ewing’s sarcoma (ES) within its bone niche. This particular strategy is based on engineered human bone by introducing osteoclasts in co-culture with osteoblasts in the 3-dimensional bone niche. This model mimics bone remodeling and recapitulates some of the features observed in the osteolytic process in cancer and also\, the effects of the therapeutic reagent Zoledronic acid observed in patients. The second strategy consists in designing biomaterials with the same tumor composition to mimic the biological and mechanical properties of tumors from patients. I have developed 3D porous collagen 1-hyaluronic acid scaffolds (Col1-HA scaffolds) for studies of tumor derivedexosomes\, which are known to be initiators of pre-metastatic niche formation in certain sites. Interestingly\, I found high levels of a critical mediator of ES growth and metastasis (EZH2) in exosomes isolated from both patients and TE model of ES. Alternatively\, we cultured TE models based on Col1-HA scaffolds into a mechanical loading bioreactor for better mimicking biomechanical forces in ES. We found that biomechanical stimuli modulate osteolytic-related proteins (i.e. RUNX2) and sensitivity to anticancer drugs\, such as Sorafenib. I will also explain the use of perfusion bioreactors and cell sheet engineering to develop a novel model of Neuroblastoma (NB) to study the effect of consolidative drugs\, such as Isotretinoin\, on tumor vasculature and stem-like cells. Here\, I will show the existence of sub-populations of NB cells with different levels of stemness properties; these levels are related to the capacity of stem-like cells to transdifferentiate and also\, to chemoresistance and relapse. Finally\, the take-home message of my talk will be that TE models can bridge the gap between 2D in vitro cultures and in vivo animal models in a predictive\, inexpensive and low timeconsuming fashion for successfully understand cancer biology and improve cancer treatments.
URL:https://ibecbarcelona.eu/event/ibec-seminar-aranzazu-villasante-3/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170329T120000
DTEND;TZID=Europe/Madrid:20170329T130000
DTSTAMP:20260406T000101
CREATED:20170308T135727Z
LAST-MODIFIED:20170308T135727Z
UID:28031-1490788800-1490792400@ibecbarcelona.eu
SUMMARY:IBEC Seminar: Aranzazu Villasante
DESCRIPTION:Cancer Engineering: Strategies to Engineer Predictable Tumor Models\nDr. Aranzazu Villasante\, Department of Biomedical Engineering\, Columbia University\, New York\nAlthough many drugs show promise in monolayer or in animal models systems\, most fail to translate in humans and this is because they lack of ability to replicate the human microenvironment in patients. In response to these limitations\, I have generated a set of predictable tissue-engineered (TE) models of cancer by using different strategies. Today\, I am going to focus on some of these approaches to engineer pediatric tumors in vitro. Firstly\, I will show a TE model of Ewing’s sarcoma (ES) within its bone niche. This particular strategy is based on engineered human bone by introducing osteoclasts in co-culture with osteoblasts in the 3-dimensional bone niche. This model mimics bone remodeling and recapitulates some of the features observed in the osteolytic process in cancer and also\, the effects of the therapeutic reagent Zoledronic acid observed in patients. The second strategy consists in designing biomaterials with the same tumor composition to mimic the biological and mechanical properties of tumors from patients. I have developed 3D porous collagen 1-hyaluronic acid scaffolds (Col1-HA scaffolds) for studies of tumor derivedexosomes\, which are known to be initiators of pre-metastatic niche formation in certain sites. Interestingly\, I found high levels of a critical mediator of ES growth and metastasis (EZH2) in exosomes isolated from both patients and TE model of ES. Alternatively\, we cultured TE models based on Col1-HA scaffolds into a mechanical loading bioreactor for better mimicking biomechanical forces in ES. We found that biomechanical stimuli modulate osteolytic-related proteins (i.e. RUNX2) and sensitivity to anticancer drugs\, such as Sorafenib. I will also explain the use of perfusion bioreactors and cell sheet engineering to develop a novel model of Neuroblastoma (NB) to study the effect of consolidative drugs\, such as Isotretinoin\, on tumor vasculature and stem-like cells. Here\, I will show the existence of sub-populations of NB cells with different levels of stemness properties; these levels are related to the capacity of stem-like cells to transdifferentiate and also\, to chemoresistance and relapse. Finally\, the take-home message of my talk will be that TE models can bridge the gap between 2D in vitro cultures and in vivo animal models in a predictive\, inexpensive and low timeconsuming fashion for successfully understand cancer biology and improve cancer treatments.
URL:https://ibecbarcelona.eu/event/ibec-seminar-aranzazu-villasante/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170329T120000
DTEND;TZID=Europe/Madrid:20170329T130000
DTSTAMP:20260406T000101
CREATED:20170308T135727Z
LAST-MODIFIED:20170308T135727Z
UID:96010-1490788800-1490792400@ibecbarcelona.eu
SUMMARY:IBEC Seminar: Aranzazu Villasante
DESCRIPTION:Cancer Engineering: Strategies to Engineer Predictable Tumor Models\nDr. Aranzazu Villasante\, Department of Biomedical Engineering\, Columbia University\, New York\nAlthough many drugs show promise in monolayer or in animal models systems\, most fail to translate in humans and this is because they lack of ability to replicate the human microenvironment in patients. In response to these limitations\, I have generated a set of predictable tissue-engineered (TE) models of cancer by using different strategies. Today\, I am going to focus on some of these approaches to engineer pediatric tumors in vitro. Firstly\, I will show a TE model of Ewing’s sarcoma (ES) within its bone niche. This particular strategy is based on engineered human bone by introducing osteoclasts in co-culture with osteoblasts in the 3-dimensional bone niche. This model mimics bone remodeling and recapitulates some of the features observed in the osteolytic process in cancer and also\, the effects of the therapeutic reagent Zoledronic acid observed in patients. The second strategy consists in designing biomaterials with the same tumor composition to mimic the biological and mechanical properties of tumors from patients. I have developed 3D porous collagen 1-hyaluronic acid scaffolds (Col1-HA scaffolds) for studies of tumor derivedexosomes\, which are known to be initiators of pre-metastatic niche formation in certain sites. Interestingly\, I found high levels of a critical mediator of ES growth and metastasis (EZH2) in exosomes isolated from both patients and TE model of ES. Alternatively\, we cultured TE models based on Col1-HA scaffolds into a mechanical loading bioreactor for better mimicking biomechanical forces in ES. We found that biomechanical stimuli modulate osteolytic-related proteins (i.e. RUNX2) and sensitivity to anticancer drugs\, such as Sorafenib. I will also explain the use of perfusion bioreactors and cell sheet engineering to develop a novel model of Neuroblastoma (NB) to study the effect of consolidative drugs\, such as Isotretinoin\, on tumor vasculature and stem-like cells. Here\, I will show the existence of sub-populations of NB cells with different levels of stemness properties; these levels are related to the capacity of stem-like cells to transdifferentiate and also\, to chemoresistance and relapse. Finally\, the take-home message of my talk will be that TE models can bridge the gap between 2D in vitro cultures and in vivo animal models in a predictive\, inexpensive and low timeconsuming fashion for successfully understand cancer biology and improve cancer treatments.
URL:https://ibecbarcelona.eu/event/ibec-seminar-aranzazu-villasante-4/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170318T110000
DTEND;TZID=Europe/Madrid:20170318T170000
DTSTAMP:20260406T000101
CREATED:20170315T095240Z
LAST-MODIFIED:20170315T095240Z
UID:96008-1489834800-1489856400@ibecbarcelona.eu
SUMMARY:IV IBEC Calçotada
DESCRIPTION:IV IBEC Calçotada\nAt “Merendero Font de les Planes”\, a picnic area where you can arrive by public transport: FGC station “Les Planes” (aprox. 20min from Barcelona). \nOrganized by the IBEC PhD Committee.
URL:https://ibecbarcelona.eu/event/iv-ibec-calcotada-2/
LOCATION:Merendero Font de les Planes
CATEGORIES:Social / Internal / PhD Committee
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170318T110000
DTEND;TZID=Europe/Madrid:20170318T170000
DTSTAMP:20260406T000101
CREATED:20170315T095240Z
LAST-MODIFIED:20170315T095240Z
UID:28168-1489834800-1489856400@ibecbarcelona.eu
SUMMARY:IV IBEC Calçotada
DESCRIPTION:IV IBEC Calçotada\nAt “Merendero Font de les Planes”\, a picnic area where you can arrive by public transport: FGC station “Les Planes” (aprox. 20min from Barcelona). \nOrganized by the IBEC PhD Committee.
URL:https://ibecbarcelona.eu/event/iv-ibec-calcotada/
LOCATION:Merendero Font de les Planes
CATEGORIES:Social / Internal / PhD Committee
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170307T090000
DTEND;TZID=Europe/Madrid:20170308T170000
DTSTAMP:20260406T000101
CREATED:20170301T084527Z
LAST-MODIFIED:20170301T084527Z
UID:96006-1488877200-1488992400@ibecbarcelona.eu
SUMMARY:X Conferencia Anual de las Plataformas Tecnológicas de Investigación Biomédica
DESCRIPTION:Los próximos días 7 y 8 de marzo\, se celebrará en Madrid\, la X Conferencia Anual de las Plataformas Tecnológicas de Investigación Biomédica: Medicamentos Innovadores\, Nanomedicina\, Tecnología Sanitaria y Mercados Biotecnológicos. \nEn la Conferencia\, se van a abordar diferentes aspectos de la Innovación en el Sistema Nacional de Salud\, desde una perspectiva nacional e internacional de colaboración público-privada\, que permita a los pacientes el acceso a los nuevos fármacos\, incorporar la medicina personalizada y las nuevas tecnologías en el tratamiento de sus enfermedades\, y de este modo\, contribuir a la sostenibilidad del SNS. \nProgramme
URL:https://ibecbarcelona.eu/event/x-conferencia-anual-de-las-plataformas-tecnologicas-de-investigacion-biomedica-2/
LOCATION:Hotel Meliá Avenida de América\, C/Juan Ignacio Luca de Tena\, 36\, Madrid\, Spain
CATEGORIES:IBEC Symposium / Conference / Congress / Workshop
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170307T090000
DTEND;TZID=Europe/Madrid:20170308T170000
DTSTAMP:20260406T000101
CREATED:20170301T084527Z
LAST-MODIFIED:20170301T084527Z
UID:27938-1488877200-1488992400@ibecbarcelona.eu
SUMMARY:X Conferencia Anual de las Plataformas Tecnológicas de Investigación Biomédica
DESCRIPTION:Los próximos días 7 y 8 de marzo\, se celebrará en Madrid\, la X Conferencia Anual de las Plataformas Tecnológicas de Investigación Biomédica: Medicamentos Innovadores\, Nanomedicina\, Tecnología Sanitaria y Mercados Biotecnológicos. \nEn la Conferencia\, se van a abordar diferentes aspectos de la Innovación en el Sistema Nacional de Salud\, desde una perspectiva nacional e internacional de colaboración público-privada\, que permita a los pacientes el acceso a los nuevos fármacos\, incorporar la medicina personalizada y las nuevas tecnologías en el tratamiento de sus enfermedades\, y de este modo\, contribuir a la sostenibilidad del SNS. \nProgramme
URL:https://ibecbarcelona.eu/event/x-conferencia-anual-de-las-plataformas-tecnologicas-de-investigacion-biomedica/
LOCATION:Hotel Meliá Avenida de América\, C/Juan Ignacio Luca de Tena\, 36\, Madrid\, Spain
CATEGORIES:IBEC Symposium / Conference / Congress / Workshop
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170306T120000
DTEND;TZID=Europe/Madrid:20170306T130000
DTSTAMP:20260406T000101
CREATED:20170222T083846Z
LAST-MODIFIED:20170222T083846Z
UID:96003-1488801600-1488805200@ibecbarcelona.eu
SUMMARY:IBEC Seminar: David Cahen
DESCRIPTION:Electron Transport across Peptides and Proteins\nProf. David Cahen\, Weizmann Institute of Science\nElectron transport (ETp)\, i.e.\, electronic conduction across peptides and proteins in a solid state–like configuration is surprisingly efficient\, and comparable to\, or at times even more efficient than via completely conjugated molecules of comparable length. Working with modified proteins and with homopeptides we find both cofactors and secondary structure to matter for ETp efficiency. An open question is if contact to the external world is the dominant factor\, or intra-protein transport. This is important\, also for electron transfer\, ET: nature regulates ET via redox chemistry\, i.e.\, injection and extraction of electrons; this is where ET and ETp are related\, because the analog in the latter is the coupling to the electrodes. In ET control over the process is achieved at the free energy price of a redox event\, but no redox process is required for ETp. This allows studying ETp via non-redox proteins\, such as rhodopsins or albumins (“dopable” proteins)\, pointing to peptides as efficient transport media; studying transport via\, including coupling to them\, can help to learn about protein ETp.
URL:https://ibecbarcelona.eu/event/ibec-seminar-david-cahen-4/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170306T120000
DTEND;TZID=Europe/Madrid:20170306T130000
DTSTAMP:20260406T000101
CREATED:20170222T083846Z
LAST-MODIFIED:20170303T081702Z
UID:27719-1488801600-1488805200@ibecbarcelona.eu
SUMMARY:IBEC Seminar: David Cahen
DESCRIPTION:Electron Transport across Peptides and Proteins\nProf. David Cahen\, Weizmann Institute of Science\nElectron transport (ETp)\, i.e.\, electronic conduction across peptides and proteins in a solid state–like configuration is surprisingly efficient\, and comparable to\, or at times even more efficient than via completely conjugated molecules of comparable length. Working with modified proteins and with homopeptides we find both cofactors and secondary structure to matter for ETp efficiency. An open question is if contact to the external world is the dominant factor\, or intra-protein transport. This is important\, also for electron transfer\, ET: nature regulates ET via redox chemistry\, i.e.\, injection and extraction of electrons; this is where ET and ETp are related\, because the analog in the latter is the coupling to the electrodes. In ET control over the process is achieved at the free energy price of a redox event\, but no redox process is required for ETp. This allows studying ETp via non-redox proteins\, such as rhodopsins or albumins (“dopable” proteins)\, pointing to peptides as efficient transport media; studying transport via\, including coupling to them\, can help to learn about protein ETp.
URL:https://ibecbarcelona.eu/event/ibec-seminar-david-cahen/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170306T120000
DTEND;TZID=Europe/Madrid:20170306T130000
DTSTAMP:20260406T000101
CREATED:20170222T083846Z
LAST-MODIFIED:20170222T083846Z
UID:96001-1488801600-1488805200@ibecbarcelona.eu
SUMMARY:IBEC Seminar: David Cahen
DESCRIPTION:Electron Transport across Peptides and Proteins\nProf. David Cahen\, Weizmann Institute of Science\nElectron transport (ETp)\, i.e.\, electronic conduction across peptides and proteins in a solid state–like configuration is surprisingly efficient\, and comparable to\, or at times even more efficient than via completely conjugated molecules of comparable length. Working with modified proteins and with homopeptides we find both cofactors and secondary structure to matter for ETp efficiency. An open question is if contact to the external world is the dominant factor\, or intra-protein transport. This is important\, also for electron transfer\, ET: nature regulates ET via redox chemistry\, i.e.\, injection and extraction of electrons; this is where ET and ETp are related\, because the analog in the latter is the coupling to the electrodes. In ET control over the process is achieved at the free energy price of a redox event\, but no redox process is required for ETp. This allows studying ETp via non-redox proteins\, such as rhodopsins or albumins (“dopable” proteins)\, pointing to peptides as efficient transport media; studying transport via\, including coupling to them\, can help to learn about protein ETp.
URL:https://ibecbarcelona.eu/event/ibec-seminar-david-cahen-2/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170306T120000
DTEND;TZID=Europe/Madrid:20170306T130000
DTSTAMP:20260406T000101
CREATED:20170222T083846Z
LAST-MODIFIED:20170222T083846Z
UID:96002-1488801600-1488805200@ibecbarcelona.eu
SUMMARY:IBEC Seminar: David Cahen
DESCRIPTION:Electron Transport across Peptides and Proteins\nProf. David Cahen\, Weizmann Institute of Science\nElectron transport (ETp)\, i.e.\, electronic conduction across peptides and proteins in a solid state–like configuration is surprisingly efficient\, and comparable to\, or at times even more efficient than via completely conjugated molecules of comparable length. Working with modified proteins and with homopeptides we find both cofactors and secondary structure to matter for ETp efficiency. An open question is if contact to the external world is the dominant factor\, or intra-protein transport. This is important\, also for electron transfer\, ET: nature regulates ET via redox chemistry\, i.e.\, injection and extraction of electrons; this is where ET and ETp are related\, because the analog in the latter is the coupling to the electrodes. In ET control over the process is achieved at the free energy price of a redox event\, but no redox process is required for ETp. This allows studying ETp via non-redox proteins\, such as rhodopsins or albumins (“dopable” proteins)\, pointing to peptides as efficient transport media; studying transport via\, including coupling to them\, can help to learn about protein ETp.
URL:https://ibecbarcelona.eu/event/ibec-seminar-david-cahen-3/
CATEGORIES:IBEC Seminar
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=Europe/Madrid:20170303T170000
DTEND;TZID=Europe/Madrid:20170303T220000
DTSTAMP:20260406T000101
CREATED:20170224T093010Z
LAST-MODIFIED:20170224T093123Z
UID:27775-1488560400-1488578400@ibecbarcelona.eu
SUMMARY:IBEC Lab Tour
DESCRIPTION:Do you ever wonder what kind of research other groups do at IBEC?\nIBEC’s PhD student committee is very pleased to invite you to the IBEC Lab Tour. \nIt’s an opportunity to visit other groups’ laboratories at IBEC and find out what they’re doing. All  are welcome regardless of position (undergraduate student\, master student\, PhD student\, postdoc\, technician\, etc.) \nAfter the tour\, there will be a pica-pica and more time for networking\, and then a visit to Bowling Pedralbes for some bowling\, beers and lots of fun. \nIf you want to join this activity\, please register in the Doodle at http://doodle.com/poll/35yikkawr332atik before Monday 27th of February.\n  \nMeet at the Helix Building Reception at 17h00
URL:https://ibecbarcelona.eu/event/ibec-lab-tour/
LOCATION:IBEC\, Baldiri Reixac 10-12\, Barcelona\, 08028\, Spain
CATEGORIES:Social / Internal / PhD Committee
END:VEVENT
END:VCALENDAR